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EXPOSURE TO AERIAL EMISSIONS OF NANO COMPOSITE MATERIALS RESULTED IN CHOLINESTERASE INHIBITION - By Toxicologist Dr. Hildy Staninger

Ana Maria Mihalcea, MD, PhD - Dec 08, 2023 ∙ Paid ∙ Source

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I am posting this paper on the effect of advanced nanotechnology particles on the Acetyl cholineesterase system. Much discussion has been held that the Covid virus, - that has not been identified anywhere - or snake venom peptides that supposedly have been ubiquitously deployed - cause effects on the nicotinic actetyl choline receptor. Hence Nicotine should be indicated either to prevent shedding - which it does not - or to improve long Covid symptoms. I would like to present this historical evidence that it has long been known that aerosolized nanoparticles are causing this inhibition also - and hence can expand our understanding of the subject.

In my clinical experience, Nicotine does not always work symptomatically, because other receptor sites are also affected. If used as a symptomatic treatment it should not be done as solo therapy. In my observation, after an initial period of improvement lasting a few weeks, symptoms returned. Recontamination with self replicating nanotechnology due to shedding or environmental exposure, can cause recurrence of symptoms while on Nicotine.

Please note we found silicone signatures in the blood and in the C19 vials. Carbon nanotubes (Graphene Oxide) are known to be in the C19 shots according to Pfizers own documents. In the article below, these symptoms are correlated with Silicone Carbon Nanotubes and other advanced nanomaterials.

The detoxification process of the nanotechnology is imperative for long term clinical improvement. Please note, I do not advocate against Nicotine, for whomever it has beneficial effects. I caution however - against believing that people are safe from shedding - because they are not. I have looked at hundreds of live blood analysis of people who were on Nicotine and have not found one that had clean blood. If used, it should be used as an adjunct symptomatic therapy while other treatments addressing the root cause of nanotechnology contamination are utilized.

EXPOSURE TO AERIAL EMISSIONS OF NANO COMPOSITE MATERIALS RESULTED IN CHOLINESTERASE INHIBITION

by Hildegarde Staninger, Ph.D., RIET-1, Industrial Toxicologist/IH & Doctor of Integrative Medicine.  Presented at the 2009 National Registry of Environmental Professionals Conference Oct 5-6 ^th , 2009, Des Plaines, IL.   Published NREP Journal of Environment and Sustainability © Oct 2009.

ABSTRACT :  Cholinesterase inhibition has been associated with exposure to organophosphate and carbamate pesticides, since their creation during World War I and II.  Traditionally, they cause severe neurological disorders that can paralyze not only insects but humans.  Individuals are usually exposed by misuse of pesticide applications or through aerial spraying.  The cholinesterase inhibition effects for chronic exposure or multiple acute exposures are expressed in acetyl cholinesterase, enzyme (AChE) activities inhibition.  The integration of nano particulates in various nano composite materials, such as aerial hydrogels and other similar materials used in aerial vector sprays, weather modification and sensor grids, resulted in the individual having as high as 96.2 % cholinesterase inhibition and detectable readings of nano composite materials present.  Confirming Ocean University, China’s findings in the June 2009 issue of Chemical Sensitivities, that nano particles are 100 times more toxic than a single molecule of a pesticide, such as Malathion, propoxopur or benomyl.

Background:

During the summer of 2007 in Phoenix, Arizona, a female age 51 was exposed to aerial spraying for weather modification and vector control (mosquito) as performed through G-1 requirements and Project Earth Scope ¹ .  She was at home and had her windows open and went outside of her home to see what was happening. She immediately felt a burning and tingling sensation and became ill.   After numerous analytical testing and physician evaluations, it was determined she was exposed at a minimum to nano composite material and Sencil™ ¹ technology due to specimens of the materials taken from her body appeared to be hair follicles, but were not upon laboratory analysis.   The sample melted at 650 ^o C, while human hair melts at just above 135 ^o C and synthetic hair melts at approximately 225 ⁰ C.

Discussion:

Primary concern was individual’s symptoms, which coincided with chronic acetyl cholinesterase (AChE) enzyme inhibition in the circulating red blood cells that are depository regulators for the various soft tissue organs within the body. ²  Once the levels of the red blood cell values become very high they trigger the release of plasma cholinesterase to replenish any future inhibition roller coasting effects.   The roller coasting effects can be very sudden or gradual; all depending upon the amount of recovery of the enzyme into the organ systems and the trigger points of specific sensor modulating factors within the cell membranes and nuclear membrane.

A comprehensive toxicological analysis of the results using the standard mathematical calculations to determine the percent (%) inhibition of acetylcholinesterase was determined from initial testing in 2008 to present 2009 values.  The values were calculated and based upon the most current cholinesterase value for red blood cells and plasma to show a linear time relationship and medically observations by her treating physician/naturopath and toxicologist from the chronic effects of cholinesterase inhibition and its toxicological neurological mechanisms of action.

On August 15, 2009 her individual value for cholinesterase, plasma 2019 and RBC 5774 IUs.   A comparative table is stated below, based on each date’s value and compared to August 15, 2008 (note exactly one year from initial testing).

Table 1 -1: Date vs. Value Level (IU) and Calculated % Inhibition of

Cholinesterase

Plasma

                                   08/15/2008                 07/21/2009                  08/11/2009        

                                     2019                             4928                            4581

% Inhibition                 Base Value (BV)               59%                              7.57%

NOTE: BV to Most Resent Test Value % Inhibition is  55.92% inhibition of Plasma Cholinesterase over a 1 year period.

RBC

                                   08/15/2008                 07/21/2009                  08/11/2009

                                     5774                           2080 L                         15,092

% Inhibition                Base Value (BV)               96.4%                         86.2%

NOTE:  BV to Most Resent Test Value % Inhibition is 61.74% inhibition of RBC Cholinesterase over a 1 year period.  Another individual exposed to nano composite and nanoparticulate via water; female age 50 had 76.2 % RBC AChe inhibition. Control male age 70  had 0.3 % RBC Ache inhibition and no exposure to advanced nano microbic materials of any type .  The control’s value is within standard deviation of <= 2% inhibition value.

The US Federal Government’s standard for chronic cholinesterase inhibition was set by the US EPA Agency in EPA Recognition and Management of Pesticide Poisonings, 4 ^th Edition, US Governmental Printing Office, Washington, D.C. March 1989 EPA-0540/9-88-001 that a 10 % or more inhibition factor is a result of chronic pesticide poisoning.  A number of advanced nano microbic materials are made from composite material that contains mixtures of dragon protein, carbamates, and microbivores each containing specific chemicals that are known to cause cholinesterase inhibition. 

The difference between a regular chemical and/or pesticide exposure and nano advanced materials is that the nano materials are able to penetrate the cell membrane and attach to the cellular membrane as G protein/C-reactive protein with chemical interactions on the nuclear membrane, which would result in measurable value of antinuclear antibody immunoglobulins. ² Because of this primary factor of entering the cytoplasma, organelles and nuclear membrane nano materials are cytotoxic and cause cellular dysfunction, injury and initiate toxicological mechanisms of disease patterns.  This is further complicated with previous exposure to childhood viruses (measles and chicken pox) and current aerial testing emissions for DARPA under DARPA’s Unconventional Pathogen Countermeasure Program and Project Earth Scope. ^{3, 4}

The individual had specific tests performed on ionic bath samples and “pseudo hair” samples, which revealed exposure to – nano composite material- that came out of her body.  This material was sent to Applied Consumer Services, Inc. for advanced materials analysis and Morris Consulting, Inc. both laboratories are governed by GPL standards within the laboratory.

Recent studies conducted by the College of Environmental Sciences and Engineering, Ocean University of China, Qingdao, China (Z. Wang, et.al.) ^5,6 h ave shown that manufactured nanoparticles can be toxic via interactions with proteins (precursor’s amino acids) and enzymes.  Acetylcholinesterase (AChE) is a key enzyme present in the brain, blood and nervous system.  Therefore, absorption and inhibition of AChE by specific nanoparticles, SiO ₂ , TiO ₂ , Al ₂ O ₃ , Al, Cu, and Cu-C (carbon coated copper), multi-walled carbon nanotubes (MWCNT) and single-walled carbon nanotubes (SWCNT) had the following results:

• Carbon nanotubes had high affinity for AChE absorption.  SWCNT (94%),

Nano SiO ₂ and Al ₂ O ₃ showed the lowest absorption.

• Cu(2+) release in Cu and Cu-C nanoparticle suspensions caused 40% and 45% of AChE activity reduction, respectively AChE inhibition by bulk Cu and

activated carbon particles were also measured for comparison, showing that the inhibition by bulk particles was lower than their counterpart nanoparticles .

• Bulk Cu particles, AChE inhibition was primarily caused by dissolved ions, but mainly absorption for activated charcoal.

• AChE inhibition by Cu, Cu-C, MWCNT and SWCNT had dose-response relationships, and their median inhibitory concentrations (IC 50) were 7, 17, 156 and 96 mg/L ^-1 , respectively, showing that these nanoparticales may have neurotoxicity and AChE may have potential to be used as a biomarker for nanoparticle exposure.

The primary toxicological mechanisms of action of the nanoparticles used in nanocomposite materials are mainly due to cholinesterase inhibition.  Three main biochemical reactions are responsible for these toxicological effects.

1. Inhibition of cholinesterase activity.

2. Inhibition of neuropathy target esterase (NTE) and development of delayed neuropathy.

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